Mitochondrial autophagy is an early defense and protection process that selectively clears dysfunctional or excessive mitochondria through a distinctive mechanism to maintain intracellular homeostasis. Mitochondrial dysfunction during cerebral stroke involves metabolic disbalance, oxidative stress, apoptosis, endoplasmic reticulum stress, and abnormal mitochondrial autophagy. This article reviews the research progress on the mechanism of mitochondrial autophagy in ischemic stroke to provide a theoretical basis for further research on mitochondrial autophagy and the treatment of ischemic stroke.
基金：
National Natural Science Foundation of ChinaNational Natural Science Foundation of China (NSFC) [82001278]; Fund for Young Doctors with the First People's Hospital of Yunnan Province [KHBS-2020-014]; Yunnan Fundamental Research Projects; Kunming Medical University for Applied Basic Research; Yunnan Provincial Science and Technology Department
第一作者机构：[1]Kunming Univ Sci & Technol, Affiliated Hosp, Kunhua Hosp, Peoples Hosp Yunnan Prov 1,Dept Neurosurg, Kunming, Yunnan, Peoples R China
共同第一作者：
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推荐引用方式(GB/T 7714)：
Lei Li,Yang Shuaifeng,Lu Xiaoyang,et al.Research Progress on the Mechanism of Mitochondrial Autophagy in Cerebral Stroke[J].FRONTIERS IN AGING NEUROSCIENCE.2021,13:doi:10.3389/fnagi.2021.698601.
APA：
Lei, Li,Yang, Shuaifeng,Lu, Xiaoyang,Zhang, Yongfa&Li, Tao.(2021).Research Progress on the Mechanism of Mitochondrial Autophagy in Cerebral Stroke.FRONTIERS IN AGING NEUROSCIENCE,13,
MLA：
Lei, Li,et al."Research Progress on the Mechanism of Mitochondrial Autophagy in Cerebral Stroke".FRONTIERS IN AGING NEUROSCIENCE 13.(2021)
